Subcutaneous Insulin Resistant Diabetes
Source: PHQandA, Message 2219.
Medical Background:
In numerous patients with diabetes mellitus, a defectin the absorption of subcutaneously injected insulin has been suspected as an explanation for diabetic instability. The common clinical characteristic of these patients is poor metabolic control when insulinis injected subcutaneously, but good metabolic control when the insulin is infused intravenously. We have used three approaches to attempt to identify patients with “subcutaneous-insulin resistance.” This syndrome is extremely rare and that misdiagnosis is common. Obesity is associated with insulin resistance, particularly when body fat has a central distribution.
However, insulin resistance also frequently occurs in apparently lean individuals. It has been proposed that these lean insulin-resistant individuals have greater amounts of body fat than lean insulin-sensitive subjects. Alternatively, their body fat distribution may be different. Obesity is associated with elevated plasma leptin levels, but some studies have suggested that insulin sensitivity is an additional determinant of circulating leptin concentrations. Accumulation of intra-abdominal fat correlates with insulin resistance, where as subcutaneous fat deposition correlates with circulating leptin levels. We conclude that the concurrent increase in these two metabolically distinct fat compartments is a major explanation for the association between insulin resistance and elevated circulating leptin concentrations in lean and obese subjects. The role of insulin resistance in metabolic diseases has received considerable attention in recent years. Insulin resistance has been suggested to be an important risk factor in the development of the metabolic syndrome, a cluster of disorders comprising glucose intolerance, dyslipidemia, hypertension, and dysfibrinolysis that is associated with type 2 diabetes and cardio vascular disease. It is evident that obesity is a risk factor for these same conditions and that this association is not only related to the degree of obesity, but also appears tobe critically dependent on body fat distribution. Thus, individuals with greater degrees of centraladiposity appear to develop this syndrome more frequently than those with a peripheral body fat distribution.
Source:
Source: PHQandA, Message 2219.
Medical Background:
In numerous patients with diabetes mellitus, a defectin the absorption of subcutaneously injected insulin has been suspected as an explanation for diabetic instability. The common clinical characteristic of these patients is poor metabolic control when insulinis injected subcutaneously, but good metabolic control when the insulin is infused intravenously. We have used three approaches to attempt to identify patients with “subcutaneous-insulin resistance.” This syndrome is extremely rare and that misdiagnosis is common. Obesity is associated with insulin resistance, particularly when body fat has a central distribution.
However, insulin resistance also frequently occurs in apparently lean individuals. It has been proposed that these lean insulin-resistant individuals have greater amounts of body fat than lean insulin-sensitive subjects. Alternatively, their body fat distribution may be different. Obesity is associated with elevated plasma leptin levels, but some studies have suggested that insulin sensitivity is an additional determinant of circulating leptin concentrations. Accumulation of intra-abdominal fat correlates with insulin resistance, where as subcutaneous fat deposition correlates with circulating leptin levels. We conclude that the concurrent increase in these two metabolically distinct fat compartments is a major explanation for the association between insulin resistance and elevated circulating leptin concentrations in lean and obese subjects. The role of insulin resistance in metabolic diseases has received considerable attention in recent years. Insulin resistance has been suggested to be an important risk factor in the development of the metabolic syndrome, a cluster of disorders comprising glucose intolerance, dyslipidemia, hypertension, and dysfibrinolysis that is associated with type 2 diabetes and cardio vascular disease. It is evident that obesity is a risk factor for these same conditions and that this association is not only related to the degree of obesity, but also appears tobe critically dependent on body fat distribution. Thus, individuals with greater degrees of centraladiposity appear to develop this syndrome more frequently than those with a peripheral body fat distribution.
Source:
- Article on “In Search for theSubcutaneous-Insulin-Resistant Syndrome”, by DS Schadeand WC Duckworth. Vol. 315:147-153 July 17, 1986 No.3,New England Journal of Medicine.
- Article on Diabetis, April, 2002, by Miriam Cnop, Melinda J. Landchild, Josep Vidal, Peter J. Havel,Negar G. Knowles, Darcy R. Carr, Feng Wang, Rebecca L.Hull, Edward J. Boyko, Barbara M. Retzlaff, Carolyn E.Walden, Robert H. Knopp, Steven E. Kahn
- Invoke and scan before, during and aftertreatment.
- General sweeping twice.
- Localized thorough sweeping on the front and backsolar plexus chakra, the liver and the pancreas.
- Energize the back solar plexus chakra and the pancreas with LWG, LWB then ordinary LWV.
- Localized thorough sweeping on the ajna chakra. Energize it with LWG, then with more of ordinary LWV. This step is important.
- Localized thorough sweeping on the lungs: front sides and back. Energize directly through the back of the lungs with LWG then LWO. Point your fingers away from the head when energizing with orange.
- Localized thorough sweeping on the front and back spleen and the meng mein chakra. Do not energize.
- Localized thorough sweeping on the kidneys alternately with LWG and LWO.
- Localized thorough sweeping on the basic and the navel chakras. Energize them with LWR. The emphasis is from step 3 to step 9.
- Localized thorough sweeping on the front and backheart chakra. Energize the back heart with less of LWG and then more of ordinary LWV.
- Localized thorough sweeping on the throat chakra, forehead chakra, crown chakra and back head minorchakra. Energize them with LWG, then with more of ordinary LWV.
- Stabilize and release projected pranic energy.
- Repeat treatment 2 times per week.
- Daily physical exercise like hour-long hikes, swimming, aerobics, dancing, cycling, etc.
- Balanced healthy nutritious diabetic diet, with plenty of fresh dark green leafy vegetables and sufficient amounts of fresh water.
- Teach the children to do daily blessings.
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